

Well reading that first result, have you even read it? It said it may effect a rare subtype and could accelerate what we already know is happening. Not cause it.
Helps to actually read it ;)
Well reading that first result, have you even read it? It said it may effect a rare subtype and could accelerate what we already know is happening. Not cause it.
Helps to actually read it ;)
Separately, there is supposedly some evidence now that Alzheimer’s is caused by a virus, or by a history of viral infections.
You got a citation for that?
As a researcher in the field, this article doesn’t really give a good explanation of what’s going on here. Seems to say he just doubted the cause but didn’t provide any other ideas. Researchers right now are thinking the amyloid beta still plays a major part, the issue is free floating amyloid beta, not the plaques. The plaques therefore would just be another symptom of this free floating amyloid beta. Also, it’s surprisingly similar to diabetes, just localized in the brain. So don’t be overweight.
They are sideview mirrors. Most people do not have them where they should be and instead have three mirrors pointing to the rear, showing basically the same thing. Why turn your head away from the front when a quick look to the mirror is safer?
Scary that asking for a citation is considered contempting by some. That doesn’t back up what the other person said at all though. There’s a huge difference between saying a virus causes AD and saying that infections worsen cognitive decline in AD. I understand not everyone is experienced in reading scientific literature, but it’s important to recognize that and not spread misinformation if you don’t understand what is being described. The closest this paper gets is to connecting two things: infections cause inflammation and inflammation contributes to AD. Both of these things have already been known for a long time. This does not mean AD is a result of a virus or viral infections.